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Fact about The Wachters Blend of Sea Vegetation:

CoQ10 shows promise for Huntington's disease

CoQ10 shows promise for Huntington's disease 

*Friday, June 22, 2012. *Research conducted by Kevin M. Biglan, MD, MPH of
the University of Rochester and his colleagues, described in the inaugural
issue of the *Journal of Huntington's
Disease<http://www.iospress.nl/journal/journal-of-huntingtons-disease/> *,
provides more evidence for the use of coenzyme Q10 (CoQ10) to retard
Huntington disease's progression. Huntington's disease is a
neurodegenerative disorder caused by a genetic error that produces
abnormal proteins in the brain's cells. Scientists believe that these
protein deposits result in oxidative stress that ultimately kills the
cells that contain them.

CoQ10, due to its support of the cells' mitochondria and its antioxidant
effect, has been investigated as a possible agent to treat Huntington's
disease. The current research evaluated 14 Huntington's disease patients
and 6 healthy controls that had been given CoQ10 in a clinical trial known
as Pre-2Care. Participants in Pre-2Care received 1200 milligrams CoQ10
daily for eight weeks and 3600 milligrams per day for the remaining 12
weeks of the study.

Stored blood samples obtained at the beginning and end of the treatment
period were analyzed for serum 8-hydroxy-2'-deoxyguanosine (8OHdG), which
has been correlated with the presence of oxidative stress in the brain's
cells and has been found to be elevated in those with Huntington's disease
and other neurologic disorders. While the Pre-2Care study had found a
reduction in Huntington's disease symptoms after treatment with CoQ10, the
current research uncovered a 20 percent reduction in 8OHdG levels in
CoQ10-treated Huntington's disease patients as well as a nonsignificant
reduction in subjects who did not have the disease. "Identifying
treatments that slow the progression or delay the onset of Huntington's
disease is a major focus of the medical community," observed Dr Biglan,
who is a neurologist at the University of Rochester Medical Center. "This
study demonstrates that 8OHdG could be an ideal marker to identify the
presence oxidative injury and whether or not treatment is having an
impact."

He noted that "While the current data can't address the use of 8OHdG as a
surrogate marker for the clinical effectiveness of antioxidants in
Huntington's disease, we've established that 8OHdG can serve as a marker
of the pharmacological activity of an intervention."

"This study supports the hypothesis that CoQ exerts antioxidant effects in
patients with Huntington's disease and therefore is a treatment that
warrants further study," he concluded. "As importantly, it has provided us
with a new method to evaluate the efficacy of potential new treatments."

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